(I get around)
I’m always interested in the physical causes and consequences of bipolar. Here’s A Trip Into Bipolar Brains – don’t worry, you won’t need a seat belt or barf bag. Want the tl;dr? Bipolar 1 people have less brain volume and higher volume in the caudate nucleus and other areas associated with reward processing and decision making. Bipolar ii have less robust white matter (white matter can’t jump?). Bipolar people have more cerebrospinal fluid (comes in handy when the beer runs out) and reduced white matter integrity (more so in bipolar ii).
A study of 68 bipolar people aged 14-25 who attempted suicide found abnormalities in the prefrontal cortex.
The brain again – although ECT affects memory, apparently it increases neurocognitive function in people with treatment resistant bipolar.
More proof that the words celebrity and bipolar are a waste of space. We will probably have to declare war or an embargo on Lil’ Kim, because she called K. Michelle a prozac popping bipolar bitch. Kim would also like Michelle to stop using her as a respirator (?) to sell stuff. Could it all get any more meaningless? Plus, I don’t think monotherapy antidepressant treatment for bipolar has caught on yet.
As usual, bipolar people have been killing people and police people have been killing bipolar people and bipolar people have been killing themselves. You don’t really want links do you?
An 18 year old talks about her experience of being bipolar at school in the UK.
I looked up Hagop Akiskal on bipolar, as recommended by my psychiatrist – who also warned me that reading him can sometimes be challenging, because the writing isn’t great. Here’s a bit about the proposed extra bipolar subtypes:
Furthermore, evidence is now compelling that hypomania in association with antidepressant treatments requires familial bipolar diathesis for bipolar disorder (bipolar III). There also exist clinical depressions superimposed on hyperthymic temperament (bipolar IV), ref erring to individuals with subthreshold hypomanic traits rather than episodes.
And he suggests that many other conditions are actually soft bipolar conditions. There’s also some interesting stuff in that paper about the history of bipolar diagnosis. It isn’t new work, but I think it’s worth a read.
My bipolar is not soft. It could beat up Lil’ Kim without breaking a sweat. So there.
I was going to tell you I’d learned a new word while reading that paper (mixity), but when I googled, it didn’t exist.
Roughghost, I think you asked about the usefulness of more subtypes; here are his words on it:
… the clinical management of affective disorders will not improve significantly until there is recognition that many, if not most, depressions presenting clinically are, at some level, bipolar. As counter-intuitive as this suggestion might be, there is increasing evidence in its support summarized in this paper.
I’m quoting this just because I liked it:
The art of clinically managing these patients goes much beyond anticonvulsant concoctions. It requires the art of caring for temperamentally restless—albeit charming— individuals with troubled lives.